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The work present actual view of acute and chronic heart failure in children. It is devoted to incidence, symptoms, diagnostics and managment of this disease mainly for student of fith and sixth year of general medicine, but can be suitable for nurses, postgradual physicians as well. The main part of the work consists in diagnostis procedure and disesaseś managment.

Kyphoscoliosis is a well-recognized cause of respiratory failure. Kyphoscoliosis is a chronic disease, occurring in 2-3% of the general population, which leads to a distorted spinal curvature and chest wall deformity. As a consequence, respiratory function is impaired due to reduced chest wall compliance and restrictive lung function pattern arises. The development of noninvasive ventilation (NIV) has made it an accepted standard modality of care at the present time for patients with chronic respiratory failure caused by kyphoscoliosis. The presented case report describes a 49-year-old female with idiopathic kyphoscoliosis who developed chronic hypercapnic respiratory failure with the need of noninvasive ventilatory support.

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Short presentation about congenital heart diseases, fetal circulation, transitorial, neonatal, normal pediatric circulation. Symptoms of congenital heart diseases are persistent unexplained central cyanosis or desaturation – even if mild initially, desaturation or cyanosis that does not improve with oxygen or ventilation, a significant persistent difference in upper and lower limb saturations, congestive heart failure (newborn, infants),cyanosis, cardiomegaly, poor growth, arrythmias, murmurs, respiratory syndrome, bronchitis. This short presentation also includes diagnosis and treatment of congenital heart diseases.

The incidence of urolithiasis is 0.1-0.5-1%.The prevalence of of urolithiasis nearby 4%, in children 5,8/100, 000 children/year, M: F ratio 2:1. Recurency only once in a lifetime in about 50%. Different urolithiasis composition and metabolic changes in 50-75% pts. Etiology is unknown. There are multifactorial nature, endogenous, exogenous causes, intranefrotic, extranefrotic creation, metabolic disorders, local factors in the urinary tract as a small diuresis, immobilization, genetic factors-polygenic effect (RTAs and cystinuria), familial clustering, the impact of welfare (climate, social factors, physical activity, occupation, gender, age, ethnic and genetic influences, food, water, pregnancy, lactation, etc.).

Presented case report documents an example of a patient with an accidentally diagnosed chronic obstructive pulmonary disease (COPD) based on clinical signs. Diagnosis was confirmed by a pneumologist using spirometry. A bronchodilation test was carried out to confirm the irreversibility of detected obstructive ventilatory impairment. The patient was given combined bronchodilator treatment with ß2-mimetics and anticholinergics to relieve symptoms, improve functional parameters and prevent subsequent exacerbations.

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Obstructive sleep apnoea (OSA) is characterized by repetitive episodes of interruption of breathing during sleep due to partial or complete collapse of pharynx. Prevalence of mild OSA is about 9-24%, clinically significant disease is present in about 4% men and 2% women. Morphology of upper airways and body weight are important factors for development of OSA. Approximately 70% of OSA patients are overweight and 40% obese patients suffer from OSA.

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Obstructive sleep apnoea (OSA) is a highly prevalent sleep disorder that affects 2% of female and 4% of male population. The major risk factors for OSA is obesity. Less common causes of OSA may be various congenital craniofacial anomalies. The presented case report has introduced an adult male patient with Crouzon syndrome (CS), rare genetic disease leading to craniofacial deformities, who was referred to sleep laboratory with the suspicion of sleep disordered breathing.

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The case report describes the hospitalization of a 26 year old male with M. Barlow. The mitral valvuloplasty was performed. However, the peroperative ultrasonographic finding documented prolapse of the anterior cusp of the mitral valve into the outflow tract of left ventricle. The surgery was supplemented with a suture of the A2-PA segments of mitral valve. The subsequent finding was satisfactory. No more serious postoperative complications were noted. The patient was admitted to outpatient care on the eighth postoperative day.

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